Drug-receptor interactions

What type of molecule when it binds to a receptor produces (increases) a response?

What about a ligand determines how much of it binds to a receptor given its concentration?

What is the relationship between the amount of receptors occupied and the size of the response?

Addition of a competetive antagonist will shift a log concentration/response curve in what direction?

A sequence of experiments to determine the relative potency of a group of structurally related compounds is a what?

What is it called when a receptor requires more than one molecule binding to be activated?

What molecule does cAMP produced by adenyly cyclase control cell functions through?

What phenomenon is seen when after a prolonged exposure to an agonist the receptor is prevented from producing its normal effect?

Three processes are involved in this phenomenon uncoupling, sequestration and last?

This phenomenon has 2 key forms for Beta adrenoreceptors, heterologous and homologous. In heterologous PKA phosophorylates the receptor but is not specific, in homologous, what enzyme phosphorylates it?

After this the binding of what protein uncouples the receptor?

Which type of this phenomenon occurs with high concentrations of ligand?

What type of receptor sometimes requires dimerization in order to autophosphorylate?

What is it called if a drug is more effective when the target is being used more frequently?

In alphabetical order what are the three types of voltage gated Ca2+ channels found in muscle?

What type of molecules are used to switch the channels between modes (0=channel not open, 1=channel opening in bursts, 2=very long openings)?