Statistics for mental health drugs

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General Stats
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General Stats

This quiz has been taken 23 times
The average score is 12 of 33

Answer Stats

Hint	Answer	% Correct
	dopamine	79%
what are the drug types used to treat depression?	SSRIs (selective serotonin reuptake inhibitors)	79%
what is an example of each of these drug types?	escitalopram, sertraline, etc	68%
	serotonin	68%
	serotonin	68%
	control of appetite, sleep, mood, hallucinations, pain perception and vomiting	63%
	SNRIs (serotonin-noradrenaline reuptake inhibitors)	63%
	noradrenaline	58%
	noradrenaline	58%
what is an example of each of these drug types?	diazepam, lorazepam, etc	53%
what neurotransmitters are anxiety symptoms mediated by?	GABA	47%
what are the main neurotransmitters of the CNS?	GABA	47%
what are the two main drug types that are used to treat anxiety?	benzodiazepines	42%
what is depression characterised by?	low mood, lack of motivation and interest, and aversion to activity	42%
	MAOIs (monoamine oxidase inhibitors)	42%
	TCAs (tricyclic antidepressants)	37%
	venlafaxine, desvenlafaxine, etc	37%
	inhibits serotonin, sigma, muscarinic, NMDA and histamine receptors	26%
what is anxiety characterised by?	a feeling of dread, apprehension or uneasiness that often arises from an unknown source	21%
	amitriptyline, amoxapine, etc	21%
	barbiturates	21%
what is their mechanism of action?	binding to allosteric site of GABA-A receptors to enhance GABA binding	21%
	blocks the reuptake of serotonin and noradrenaline back into the pre-synaptic cell	21%
	blocks the reuptake of serotonin and noradrenaline back into the pre-synaptic cell, meaning that both neurotransmitters stay in the synaptic gap longer and may bind again to their receptors	21%
	phenobarbitone, amobarbitone, etc	21%
	pirlindole, selegiline, etc	21%
what is their mechanism of action?	selectively blocks the reuptake of serotonin back into the pre-synaptic cell, meaning that free serotonin stays in the synaptic gap longer and may bind again to serotonin receptors	21%
	increases arousal and alertness	16%
what is thought to be the pathophysiology of depression?	insufficient monoamine neurotransmitter activity	16%
what is thought to be the pathophysiology of anxiety?	involves neurochemical dysfunction (e.g. decreased GABA-ergic tone, increased cortisol expression) and/or dysfunction of the amygdala in the brain	11%
what is the function of each in the CNS?	mediates majority of inhibitory post-synaptic potentials	11%
what is the aims of pharmacological treatment of depression?	restore monoamine neurotransmitter activity	11%
	inhibits the activity of monoamine oxidase A and/or B, preventing the enzymes from breaking down monoamine neurotransmitters, increasing their availability	5%