Statistics for cardiovascular drugs

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General Stats
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General Stats

This quiz has been taken 21 times
The average score is 26 of 68

Answer Stats

Hint	Answer	% Correct
What are the positive inotropes?	Digoxin	75%
	Digoxin	75%
What are the negative chronotropes?	Digoxin	75%
	Beta blockers	69%
	calcium channel blockers	63%
	calcium channel blockers	63%
What are the negative inotropes?	calcium channel blockers	63%
	Calcium channel blockers	63%
	Dobutamine	63%
	Amiodarone	56%
	Atenolol	56%
	Atenolol	56%
	Atenolol	56%
What are the vasodilators?	Angiotensin Converting Enzyme (ACE) inhibitors	56%
What are the diuretics?	loop diuretics (renal diuretic)	56%
What are examples of each?	Perindopril (and other -prils)	56%
	potassium channel blockers	56%
	potassium sparing diuretics (renal diuretic)	56%
What type of blocker is each?	sodium channel blockers	56%
	used on occasion in preference to frusemide if concerned about potassium levels	56%
	Amlodipine	50%
What are examples of these?	Amlodipine	50%
	cerebral oedema (after head trauma)	50%
What are examples of these?	Frusemide	50%
	Irbesartan (and other -sartans)	50%
	Mannitol	50%
	Nitrates	50%
	osmotic diuretics	50%
	Spironolactone	50%
What are the antiarrhythmics?	Class I	44%
	Class II	44%
	Class III	44%
	Class IV	44%
What are the clinical indications for vasodilators?	heart failure	44%
	Diltiazem	38%
	Diltiazem	38%
	glaucoma	38%
What is an example of each?	Lignocaine	38%
	Sotalol	38%
	aldosterone antagonist (aldosterone drives the Na+-K+- ATPase pump)	31%
	hypertension	31%
	Nitroglycerin	31%
	Prazosin	31%
What are the common uses?	to improve myocardial contractility	31%
	β1 antagonism leading to decreased heart rate and negative inotropy, leading to decrease blood pressure	25%
What are they used for?	fluid accumulation in the lungs, abdomen, or pleural space after heart failure	25%
	Nitroprusside	25%
	Propranolol	25%
	Propranolol	25%
	to prevent arrhythmias	25%
	increases plasma osmolality so moves fluid from tissues to plasma through osmosis	19%
	used as a prodrug that converts to nitric oxide, leading to vasodilation & reduced preload	19%
	used to improve myocardial contraction force to improve cardiac output	19%
	Angiotensin II receptor inhibitors	13%
	blockade of sympathetic autonomic effects	13%
What will the use of vasodilators do?	decrease afterload	13%
	improve peripheral perfusion	13%
What is the mechanism of action of each?	Na+-K+-2Cl- pump antagonist, therefore creating an enormous natriuresis (sodium in urine) and subsequent diuresis	13%
	acts as an antagonist at the Angiotensin II receptor, therefore inhibiting the production of ADH and Aldosterone	6%
	blocks the influx of calcium into cardiac and vascular smooth muscle cells, therefore decreasing myocardial contractility and causing vasodilation	6%
	Class II antiarrhythmics	6%
	Class II antiarrythmics	6%
What is the mechanism of action for each?	inhibits the Angiotensin Converting Enzyme, therefore inhibiting the production of Angiotensin II	6%
What kind of agonists/antagonists are they?	Na+ - K+ - ATPase channel antagonist	6%
	Na+ - K+ - ATPase channel blocker	6%
	β1 agonist	0%
	α1 antagonists	0%
	α1 selective antagonist, leading to vasodilation	0%